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NCSU Extension Swine Husbandry A more printable version of Swine News in Adobe Acrobat. ![]()
UNDERSTANDING PORK QUALITY
Introduction Pork quality is one of the main issues of concern for the pork industry in the U.S. For pork to be the meat of choice, meat quality problems associated with Porcine Stress Syndrome (PSS) need to be addressed. Sonka et al. (1994) reported in the Pork Chain Quality Audit that total cost per pig from color and Pale Soft and Exudative meat (PSE) related problems were $1.05. Of this amount, $0.79 per pig was reported to be directly controllable by pig producers. In an industry that produces 88 million swine annually, this amounts to a total of $69.5 million in losses due to pork quality problems. These numbers may be an underestimate considering that some pork processing facilities reported that up to 40% of the pigs they process have PSE related problems during the summer (Morgan et al., 1994). The main quality concerns identified by all members of the pork marketing chain are poor color and inadequate waterholding capacity (Cannon et al., 1995). Therefore, addressing these quality traits is of great priority to increase the market value of pork and to provide consumers with a desirable product. Pork Quality
Measurements Pork color is mainly determined by the chemical
state of the muscle pigment myoglobin. Myoglobin consists of a heamatin portion
which contains a central iron atom and a globulin type protein. The status of
the iron (ferrous or ferric) in the heamatin portion and the status of the
globin will determine the color of the pigment. In fresh pork, the myoglobin
exists in an oxygenated form (oxymyoglobin) which results in a bright red
color. A purplish-red color is associated with myoglobin that is not
oxygenated. Oxidation of myoglobin or oxymyoglobin to metmyoglobin is
responsible for a brown color. The color of pork can be significantly influenced by the pH of the meat and the chilling process after slaughter. In the case of a high ultimate pH, residual enzyme activity will be high, resulting in removal of oxygen from the oxymyoglobin molecule. Muscle fibers will also be packed tightly together because the waterholding capacity of proteins above the iso-electric point (as is the case with high pH) is greater than below the iso-electric point. This results in dark and firm meat (DFD). In the case of PSE, a fast drop in pH or a low ultimate pH favor the oxidation of myoglobin to metmyoglobin, which results in a pale appearance (Lawrie, 1985). The importance of pH in the development of PSE, DFD, and other quality categories of pork is illustrated in Figure 1. ![]() Figure 1. Effect of initial and ultimate pH on pork quality (from PIC, 1997)
Post-mortem glycolysis is responsible for the drop in pH. Glycogen stores in muscle are converted into glucose which is subsequently converted to lactic acid in the absence of oxygen. If glycogen stores are high and the temperature of the carcass is relatively high, glycolysis will proceed at a fast rate and the ultimate pH will be low. Glycolysis will cease either because glycogen stores are depleted or the pH has reached a point at which enzymes become inactivated, typically at a pH of about 5.5 (Lawrie, 1985). The color of meat can be scored subjectively by comparing the meat color to a standardized color chart. The NPPC (1999) has recently changed their 5 color standard system to a 6 standard system, which concurs with the Japanese scale. The optimal color is 3 to 4, which represents a reddish pink to a dark reddish pink pork color. More objectively, color can be measured using a Minolta Chromameter. This method measures the brightness (L* value), the redness (a* value), and the yellowness (b* value) of the sample. The NPPC (1999) suggested that the L values for their range of light color quality pork (color standard 1) to dark quality pork (color standard 6) should be approximately 61, 55, 49, 43, 37, and 31, respectively. Thus, the ideal L* value would appear to be between 43 and 49. Drip loss from fresh pork is a result of shrinkage of muscle proteins (actin and myosin) and the subsequent expressing of fluids from the meat. At a low pH, the ability of proteins to bind water is diminished which will increase drip loss. In addition, a fast drop in pH will cause the actin-myosin complex that is formed during rigor mortis to contract and expel more fluid from the meat (Lawrie, 1985). The importance of pH in the water holding capacity, and the development of dry or exudative pork is illustrated in Figure 1. Drip loss can be measured by placing a pre-weighed filter paper on the muscle surface for a few seconds after which water uptake by the filter paper is determined. True drip loss can be measured by hanging portions of pork muscle of known weight suspended above a surface for approximately 48 hours at 4 °C. The sections of pork muscle are then weighted and the loss in weight as a fraction of the initial weight is a measure of drip loss. The oxidative status of meat is often measured by determining the thiobarbituric acid reactive substances (TBARS). Malonaldehyde, a breakdown product of oxidized fatty acids is measured through its reaction with TBA (2-thiobarbituric acid) which results in a primarily red product. In addition, TBA can react with other substances and can thus be useful in determining oxidative spoilage from compounds other than fatty acids to give an estimate of overall oxidative stability. A level of approximately 1.0 mg MDA per kg tissue has been suggested as the threshold level in which rancidity or warmed-over flavor could be detected by a taste panel (Gray and Pearson, 1987). Standard measurements of meat quality, their ranges, and the optimum scores for each measurement are summarized in Table 1. The resulting pork quality categories most commonly encountered are listed in Table 2.
Table 1. Measures of meat quality (adapted from PIC, 1997)
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Figure 2. Categories of pork quality (adpated from PIC, 1997)
![]() Literature Cited Cannon, J. E., J. B. Morgan, F. K. McKeith, G. C. Smith, S. Sonka, J. Heavner, and D. L. Meeker. 1995. Pork chain quality audit packer survey: Quantification of pork quality characteristics. J. Muscle Foods: 6:369. Gray, J. I., and A. M. Pearson. 1987. Rancidity and warmed-over flavour. Adv. Meat Res. 3:221. Lawrie, R. A. 1985. Meat Science. 4th Edition. Pergamon Press, Oxford, UK. Morgan, J. B., G. C. Smith, J. Cannon, F. McKeith, and J. Heavner. 1994. Pork distribution channel audit report. Pork Chain Quality Audit, Progress Report, April 6, 1994. p. 30. NPPC, 1999. Pork Quality Standards. National Pork Producers Council, Des Moines, IA. PIC, 1997. Meat Quality. Understanding industry measurements and guidelines. PIC, Spring, 1997, Franklin, KY. Sonka, S., T. Doehring, S. Hofing. 1994. An economic assessment of enhancing quality management in the pork industry. Pork Chain Quality Audit, Progress Report, April 6,1994. p. 170. Eric van Heugten
At the time of writing (March 14, 2001) Europe, and Britain in particular, are in the midst of an outbreak of Foot-and-Mouth Disease (FMD). The first case was detected at a slaughter plant on February 27, 2001 and by today the total confirmed cases in Great Britain is 214 with no sign of abating. This rapid spread is typical of FMD and serves as a warning to everyone in the swine industry. Given the tight security in Britain, FMD is unlikely to enter the US; however, it could. We recommend that pork producers review their biosecurity procedures. Until the crisis is over, it would be prudent to disallow any farm visits by people who have visited Europe since February, 2001 and, as always, ensure no human food scraps are fed to pigs. Specifically, the North Carolina Department
of Agriculture and Consumer Services is recommending the following precautions as an aid in
preventing introduction:
"Livestock owners are asked to be especially observant of the health status of thier animals and investigate any animal showing clinical signs suggestive of FMD. Signs include blisters or sores on the muzzle, feet, teats, or mouth; lameness associated with foot lesions; or lack of appetite or excessive slobbering associated with mouth lesions.
While other diseases can cause similar clinical signs, anything suspicious should be pursued through veterinary examination. Producers and veterinarians are encouraged to report any suspicious case to the NCDA&CS, Veterinary Division at 919-733-7601." The reason that FMD spreads so quickly is that it is one of the most contagious animal diseases, is resistant to common disinfectants, and can persist in contaminated fodder and the environment for up to one month, depending on the temperature and pH conditions. The FMD virus is preserved by refrigeration and freezing but progressively inactivated by temperatrues above 122oF. It is inactivated by caustic soda, i.e., sodium hydroxide (2%), but is resistant to iodophores, qaternary ammonium compounds, hypoclorite and phenol, especially in the presence of organic matter. Given its rapid spread, we recommend that all herd additions be first placed in quarantine and watched very carefully for signs of disease.
Essentially, all cloven-footed animals are susceptible. That is, as well as pigs, FMD can infect cattle, zebus, buffaloes, sheep, goats, and all wild ruminants including deer. Although it can rapidly infect many animals it kills very few adults but the mortality of the young can be high due to heart disease. In addition to the vesicles (blisters) pigs may develop severe foot lesions particularly when housed on concrete and piglet mortality can be high.
FMD can be transmitted either directly by infected animals or indirectly through contaminated inanimate vectors such as vehicles and farm implements. In temperate zones, such as North Carolina, FMD can be carried on the wind as far as 40 miles overland and 190 miles over water.
Humans can mechanically carry FMD virus under their fingernails and in their noses for up to 2 days after contact with infected swine. Also, humans can be infected by handling vesicular lesions and, very rarely, show mild clinical lesions sometimes exhibiting vesicles.
FMD virus comes for the breath, saliva, feces, urine, milk, and semen of affected animals and the meat and by-products in which pH has remained above 6.0.
Even though the current European outbreak is receiving all the attention, remember, FMD is already on our doorstep. It is currently present in many Middle and South American countries including Argentina, Belize, Brazil, Bolivia, Colombia, Ecuador, El Salvador, Guatemala, Nicaragua, Paraguay, Peru, Uruguay, and Venezuela.
The effect of an FMD outbreak in the US would be economically devastating. Economists have estimated that the direct costs of an FMD outbreak could reach $10 billion in the first year with indirect costs 10 times that amount.
Review your biosecurity procedures today and help prevent the spread of all infectious diseases including FMD shoud it make it to the North American continent.
For photographs of clinical signs and more information about FMD point your web browser to http://mark.asci.ncsu.edu or contact: USDA, APHIS, Veterinary Services; Emergency Programs; 4700 River Road, Unit 41; Riverdale, MD 20737-1231; Telephone 301-734-8073; Fax 301-734-7817; http://www.aphis.usda.gov
Morgan Morrow
PORK INDUSTRY HANDBOOK CD-ROM The Pork Industry Handbook is now available on CD-ROM. The CD includes 143 fact sheets developed by various authors selected on the basis of their expertise in subject matter areas. In this format the Pork Industry Handbook can be easily searched and many of the fact sheets utilize color photos, video clips, www links and spreadsheets. All phases of pork production and marketing are covered in this project, which is coordinated on a national bases in an attmept to supply uniform information. Purchase of the CD includes updates for a three-year period. You can print an order form from our website at http://mark.asci.ncsu.edu/Publications/porkindhandbook/porkindhandbook.htm or contact your local county livestock agent or e-mail your name and address (or fax number) to Carla_McKinney@ncsu.edu and she will send a from to you.
Todd See
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Last modified March 17, 2001.
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